INT261835

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Context Info
Confidence 0.01
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.32
Pain Relevance 0.66

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytoplasm (Camk2a, Prkca) plasma membrane (Camk2a, Prkca) kinase activity (Camk2a, Prkca)
mitochondrion (Prkca) endoplasmic reticulum (Prkca) enzyme binding (Prkca)
Camk2a (Mus musculus)
Prkca (Mus musculus)
Pain Link Frequency Relevance Heat
Kinase C 10 100.00 Very High Very High Very High
nociceptor 46 73.80 Quite High
agonist 32 68.48 Quite High
Nerve growth factor 6 68.28 Quite High
bradykinin 5 67.32 Quite High
antagonist 37 67.20 Quite High
Inflammatory mediators 1 64.28 Quite High
Glutamate 6 61.40 Quite High
Pain 92 52.36 Quite High
qutenza 23 50.00 Quite Low
Disease Link Frequency Relevance Heat
Nociception 17 79.04 Quite High
Disease 17 71.84 Quite High
INFLAMMATION 14 63.88 Quite High
Pain 96 52.36 Quite High
Hyperalgesia 27 5.00 Very Low Very Low Very Low
Targeted Disruption 23 5.00 Very Low Very Low Very Low
Neuropathic Pain 21 5.00 Very Low Very Low Very Low
Headache 6 5.00 Very Low Very Low Very Low
Nervous System Injury 5 5.00 Very Low Very Low Very Low
Stress 4 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Other pathways that potentiate TRPV1 are activation by PI3K [244] either via calmodulin-dependent kinase II (CaMKII), PKC, or ERK [244] or PKA [185].
CaMKII Positive_regulation (activation) of PKC associated with kinase c
1) Confidence 0.01 Published 2008 Journal Current Neuropharmacology Section Body Doc Link PMC2645548 Disease Relevance 0.32 Pain Relevance 0.66

General Comments

This test has worked.

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