INT261839

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Context Info
Confidence 0.03
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0.45

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (Car2, Trpv1) transport (Trpv1) lyase activity (Car2)
transmembrane transport (Trpv1) lipid metabolic process (Trpv1) cytoplasm (Car2)
Car2 (Mus musculus)
Trpv1 (Mus musculus)
Pain Link Frequency Relevance Heat
qutenza 23 99.28 Very High Very High Very High
Kinase C 10 76.08 Quite High
agonist 32 66.40 Quite High
nociceptor 46 50.00 Quite Low
addiction 3 31.04 Quite Low
Pain 92 29.76 Quite Low
analgesia 10 29.00 Quite Low
antagonist 37 15.60 Low Low
Hyperalgesia 16 5.00 Very Low Very Low Very Low
Inflammation 15 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Pain 96 29.76 Quite Low
Disease 17 9.88 Low Low
Nociception 17 6.44 Low Low
Hyperalgesia 27 5.00 Very Low Very Low Very Low
Targeted Disruption 23 5.00 Very Low Very Low Very Low
Neuropathic Pain 21 5.00 Very Low Very Low Very Low
INFLAMMATION 14 5.00 Very Low Very Low Very Low
Headache 6 5.00 Very Low Very Low Very Low
Nervous System Injury 5 5.00 Very Low Very Low Very Low
Stress 4 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Interestingly, calcium entry following capsaicin application is found to form a CaM/Ca2+ complex with a 35-aa segment of TRPV1 and cause desensitization [154].
Ca2 Binding (complex) of TRPV1 associated with qutenza
1) Confidence 0.03 Published 2008 Journal Current Neuropharmacology Section Body Doc Link PMC2645548 Disease Relevance 0 Pain Relevance 0.45

General Comments

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