INT262504

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Context Info
Confidence 0.25
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.43
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transmembrane transporter activity (Aqp1) plasma membrane (Aqp1) nucleus (Aqp1)
cytoplasm (Aqp1)
Aqp1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Central nervous system 13 5.00 Very Low Very Low Very Low
ischemia 4 5.00 Very Low Very Low Very Low
Inflammation 3 5.00 Very Low Very Low Very Low
potassium channel 3 5.00 Very Low Very Low Very Low
nMDA receptor 3 5.00 Very Low Very Low Very Low
Glutamate receptor 3 5.00 Very Low Very Low Very Low
Dopamine 3 5.00 Very Low Very Low Very Low
midbrain 2 5.00 Very Low Very Low Very Low
Neurotransmitter 2 5.00 Very Low Very Low Very Low
addiction 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Stress 11 90.52 High High
Apoptosis 165 89.56 High High
Toxicity 2 43.16 Quite Low
Neurodegenerative Disease 14 25.52 Quite Low
Cancer 3 9.76 Low Low
Death 31 5.00 Very Low Very Low Very Low
Disease 17 5.00 Very Low Very Low Very Low
Necrosis 8 5.00 Very Low Very Low Very Low
Pressure And Volume Under Development 8 5.00 Very Low Very Low Very Low
Hypoxia 6 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
While the increase in the half-life of the AQP1 protein might be in direct relation to the amount of water loss from the cell, it is also possible that it is a compensatory mechanism by the cell to allow for increased water uptake via the bidirectional channel to rectify the osmotic stress.
Positive_regulation (increase) of AQP1 associated with stress
1) Confidence 0.25 Published 2008 Journal Current Neuropharmacology Section Body Doc Link PMC2647147 Disease Relevance 0.43 Pain Relevance 0

General Comments

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