INT262587

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.02
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.44
Pain Relevance 0.03

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

isomerase activity (Renbp)
Renbp (Mus musculus)
Pain Link Frequency Relevance Heat
diabetic neuropathy 1 58.32 Quite High
Kinase C 1 48.16 Quite Low
rheumatoid arthritis 1 8.16 Low Low
Glutamate 29 5.00 Very Low Very Low Very Low
ischemia 12 5.00 Very Low Very Low Very Low
nMDA receptor 9 5.00 Very Low Very Low Very Low
Glutamate receptor 6 5.00 Very Low Very Low Very Low
antagonist 5 5.00 Very Low Very Low Very Low
Neurotransmitter 5 5.00 Very Low Very Low Very Low
Central nervous system 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Diabetes Mellitus 13 87.64 High High
Aging 4 86.24 High High
Age-related Macular Degeneration 8 61.68 Quite High
Diabetic Retinopathy 6 59.20 Quite High
Diabetic Neuropathy 1 58.32 Quite High
Neurodegenerative Disease 5 50.00 Quite Low
Stress 23 30.08 Quite Low
Death 24 25.96 Quite Low
Hypoxia 6 13.52 Low Low
Disease Progression 3 12.48 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Since AGEs are constantly forming under physiological conditions, complex receptor systems have evolved to remove senescent, glycation modified molecules and/or degrade existing AGE crosslinksfrom tissues thereby limiting their deleterious effects.
Protein_catabolism (degrade) of AGE
1) Confidence 0.02 Published 2008 Journal Current Neuropharmacology Section Body Doc Link PMC2647152 Disease Relevance 0.44 Pain Relevance 0.03

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox