INT263365

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Context Info
Confidence 0.03
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.18
Pain Relevance 0.17

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (Dlat) oxidoreductase activity (Glyr1) nucleus (Glyr1)
transferase activity, transferring acyl groups (Dlat) DNA binding (Glyr1)
Glyr1 (Rattus norvegicus)
Dlat (Rattus norvegicus)
Pain Link Frequency Relevance Heat
spinal dorsal horn 17 90.52 High High
Pain 6 86.00 High High
Glutamate receptor 1 70.04 Quite High
Spinal cord 11 50.00 Quite Low
Pyramidal cell 2 46.32 Quite Low
Hippocampus 11 39.64 Quite Low
antagonist 3 17.44 Low Low
Neuronal excitability 4 5.00 Very Low Very Low Very Low
GABAergic 3 5.00 Very Low Very Low Very Low
IPN 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Nociception 4 96.08 Very High Very High Very High
Pain 5 86.00 High High
Premenstrual Syndrome 6 5.00 Very Low Very Low Very Low
Epilepsy 2 5.00 Very Low Very Low Very Low
Anxiety Disorder 2 5.00 Very Low Very Low Very Low
Disease 2 5.00 Very Low Very Low Very Low
Inflammatory Pain 2 5.00 Very Low Very Low Very Low
Decapitation 1 5.00 Very Low Very Low Very Low
Affective Disorder 1 5.00 Very Low Very Low Very Low
Convulsion 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Thus, in addition to the well-known signaling pathways of E2 via ERs, the GlyR may be an additional central target of E2.
GlyR Regulation (target) of E2
1) Confidence 0.03 Published 2009 Journal Mol Pain Section Body Doc Link PMC2651124 Disease Relevance 0.18 Pain Relevance 0.17

General Comments

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