INT264297

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Context Info
Confidence 0.36
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0.44
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (NOL3) intracellular (NOL3) cytoplasm (NOL3)
cytosol (NOL3) extracellular space (IGF1) signal transduction (IGF1)
IGF1 (Homo sapiens)
NOL3 (Homo sapiens)
Pain Link Frequency Relevance Heat
rheumatoid arthritis 54 50.00 Quite Low
Inflammation 36 12.68 Low Low
Infliximab 8 5.00 Very Low Very Low Very Low
cytokine 6 5.00 Very Low Very Low Very Low
methotrexate 4 5.00 Very Low Very Low Very Low
visual analogue scale 2 5.00 Very Low Very Low Very Low
cINOD 2 5.00 Very Low Very Low Very Low
Inflammatory marker 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Osteoporosis 16 74.40 Quite High
Obesity 14 72.24 Quite High
Nicotine Addiction 8 71.60 Quite High
Rheumatoid Arthritis 54 50.00 Quite Low
Endometriosis (extended) 22 32.40 Quite Low
INFLAMMATION 40 12.68 Low Low
Disease 38 5.00 Very Low Very Low Very Low
Hypercalcemia 34 5.00 Very Low Very Low Very Low
Insulin Resistance 4 5.00 Very Low Very Low Very Low
Arthritis 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In the NoP-group, a higher IGF-1 at 12 months was associated with a more pronounced decrease in 1CTP between 0 and 12 months (r = -0.25, p = 0.034).


IGF-1 Binding (associated) of NoP
1) Confidence 0.36 Published 2008 Journal Arthritis Res Ther Section Body Doc Link PMC2656227 Disease Relevance 0.22 Pain Relevance 0
In the NoP-group, a higher IGF-1 at 12 months was associated with a more pronounced decrease in 1CTP between 0 and 12 months (r = -0.25, p = 0.034).


IGF-1 Binding (associated) of NoP
2) Confidence 0.36 Published 2008 Journal Arthritis Res Ther Section Body Doc Link PMC2656227 Disease Relevance 0.22 Pain Relevance 0

General Comments

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