INT265584

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Context Info
Confidence 0.20
First Reported 2009
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 2
Disease Relevance 1.49
Pain Relevance 0.15

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleus (Msc) DNA binding (Msc)
Anatomy Link Frequency
astrocytes 2
Msc (Mus musculus)
Pain Link Frequency Relevance Heat
Central nervous system 9 83.36 Quite High
ischemia 5 70.08 Quite High
Spinal cord 1 69.28 Quite High
cva 1 69.04 Quite High
imagery 12 22.40 Low Low
isoflurane 6 5.00 Very Low Very Low Very Low
cytokine 3 5.00 Very Low Very Low Very Low
anesthesia 3 5.00 Very Low Very Low Very Low
metalloproteinase 3 5.00 Very Low Very Low Very Low
chemokine 3 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Cancer 200 100.00 Very High Very High Very High
Necrosis 1 94.64 High High
Hypoxia 1 90.92 High High
Stroke 27 83.88 Quite High
Cv General 4 Under Development 1 70.80 Quite High
Hemorrhage 1 69.04 Quite High
Injury 4 47.92 Quite Low
Middle Cerebral Artery Infarction 17 13.12 Low Low
Breast Cancer 7 5.00 Very Low Very Low Very Low
Aggression 6 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The concomitant MSC induced reduction of PAI-1 and increase of tPA in astrocytes of the IBZ thereby amplify tPA activity which contributes to neurite outgrowth.
Positive_regulation (induced) of Localization (reduction) of MSC in astrocytes
1) Confidence 0.20 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2815778 Disease Relevance 0.72 Pain Relevance 0.15
Growth factors that support tumor cell growth are secreted by both MSC and the tumor cells when reciprocally stimulated by one another.
Positive_regulation (stimulated) of Localization (secreted) of MSC associated with cancer
2) Confidence 0.18 Published 2009 Journal PLoS ONE Section Body Doc Link PMC2661372 Disease Relevance 0.77 Pain Relevance 0

General Comments

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