INT265754

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Context Info
Confidence 0.03
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 3
Disease Relevance 0.07
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular space (FLT1, KLKB1) extracellular region (FLT1, KLKB1) plasma membrane (FLT1, KLKB1)
cell differentiation (FLT1) endosome (FLT1) Golgi apparatus (FLT1)
Anatomy Link Frequency
plasma 3
FLT1 (Homo sapiens)
KLKB1 (Homo sapiens)
Pain Link Frequency Relevance Heat
metalloproteinase 9 7.72 Low Low
addiction 9 5.00 Very Low Very Low Very Low
antagonist 9 5.00 Very Low Very Low Very Low
ischemia 6 5.00 Very Low Very Low Very Low
Restless leg syndrome 3 5.00 Very Low Very Low Very Low
Central nervous system 3 5.00 Very Low Very Low Very Low
cytokine 3 5.00 Very Low Very Low Very Low
Inflammation 3 5.00 Very Low Very Low Very Low
tolerance 3 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Increased Venous Pressure Under Development 6 67.68 Quite High
Disease 21 38.08 Quite Low
Peripheral Arterial Disease 54 11.48 Low Low
Coronary Artery Disease 18 5.00 Very Low Very Low Very Low
Cancer 15 5.00 Very Low Very Low Very Low
Atherosclerosis 12 5.00 Very Low Very Low Very Low
Diabetes Mellitus 12 5.00 Very Low Very Low Very Low
Eclampsia 9 5.00 Very Low Very Low Very Low
Breast Cancer 6 5.00 Very Low Very Low Very Low
Leukemia 6 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The much lower complexed fractions from experimental data may indicate that in vivo, other soluble receptors (e.g., sVEGFR2, sNRP1, plasma fibronectin) can strongly compete with sVEGFR1 as plasma reservoirs for VEGF, or significant quantities of other ligands (e.g., PlGF, VEGF-B) are present to compete with VEGF for sVEGFR1 binding.
sVEGFR1 Binding (compete) of plasma in plasma
1) Confidence 0.03 Published 2009 Journal PLoS ONE Section Body Doc Link PMC2663039 Disease Relevance 0 Pain Relevance 0
While steady-state analyses showed that plasma concentrations of sVEGFR1 and VEGF could vary up to 164 pM and 2.4 pM respectively (>150% about controls) over the physiological range of kL, we further examined whether concentration ranges of these magnitudes were attainable within physiological time-course.

6.3.

sVEGFR1 Binding (concentrations) of plasma in plasma
2) Confidence 0.03 Published 2009 Journal PLoS ONE Section Body Doc Link PMC2663039 Disease Relevance 0.07 Pain Relevance 0
Significant complexed fractions of VEGF and sVEGFR1 in plasma necessitate re-evaluation of assay specificity
sVEGFR1 Binding (complexed) of plasma in plasma
3) Confidence 0.03 Published 2009 Journal PLoS ONE Section Body Doc Link PMC2663039 Disease Relevance 0 Pain Relevance 0

General Comments

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