INT267250

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Context Info
Confidence 0.08
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 1.31
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

phosphatase activity (Ptprj) plasma membrane (Ptprj)
Ptprj (Mus musculus)
Pain Link Frequency Relevance Heat
Paracetamol 2 5.00 Very Low Very Low Very Low
aspirin 2 5.00 Very Low Very Low Very Low
Central nervous system 2 5.00 Very Low Very Low Very Low
Antinociceptive 2 5.00 Very Low Very Low Very Low
imagery 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apoptosis 74 99.96 Very High Very High Very High
Death 60 96.16 Very High Very High Very High
Cancer 26 50.00 Quite Low
Targeted Disruption 16 18.92 Low Low
Lung Cancer 4 11.60 Low Low
Toxicity 8 5.00 Very Low Very Low Very Low
Leukemia 6 5.00 Very Low Very Low Very Low
Skin Cancer 4 5.00 Very Low Very Low Very Low
Reprotox - General 1 2 5.00 Very Low Very Low Very Low
Neuroblastoma 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
BE/cholesterol induced apoptosis is mechanistically related to BetA induced apoptosis
Positive_regulation (induced) of BetA associated with apoptosis
1) Confidence 0.08 Published 2009 Journal PLoS ONE Section Body Doc Link PMC2671171 Disease Relevance 0.89 Pain Relevance 0
This difference between BetA and BE/cholesterol is even more remarkable when considering the time dependency of cytotoxicity of both molecules: For BetA the maximum effect requires around 48–72 hours and a dose of 7.5–10 µg/ml (Figure 1A and 1B), while BE/cholesterol induced death is already maximum at 24 hours.
Positive_regulation (requires) of BetA associated with death
2) Confidence 0.07 Published 2009 Journal PLoS ONE Section Body Doc Link PMC2671171 Disease Relevance 0.41 Pain Relevance 0

General Comments

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