INT267255

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Context Info
Confidence 0.04
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.63
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

phosphatase activity (Ptprj) mitochondrion (Bcl2) cell morphogenesis (Bcl2)
endoplasmic reticulum (Bcl2) intracellular (Bcl2) cytoplasm (Bcl2)
Bcl2 (Mus musculus)
Ptprj (Mus musculus)
Pain Link Frequency Relevance Heat
Paracetamol 1 5.00 Very Low Very Low Very Low
aspirin 1 5.00 Very Low Very Low Very Low
Central nervous system 1 5.00 Very Low Very Low Very Low
Antinociceptive 1 5.00 Very Low Very Low Very Low
imagery 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apoptosis 37 99.96 Very High Very High Very High
Cancer 13 67.44 Quite High
Death 30 49.04 Quite Low
Targeted Disruption 8 41.28 Quite Low
Toxicity 4 5.00 Very Low Very Low Very Low
Leukemia 3 5.00 Very Low Very Low Very Low
Lung Cancer 2 5.00 Very Low Very Low Very Low
Skin Cancer 2 5.00 Very Low Very Low Very Low
Reprotox - General 1 1 5.00 Very Low Very Low Very Low
Neuroblastoma 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
We previously showed that Bcl-2 over-expression delayed BetA-induced apoptosis[17], but curiously in the case of BE/cholesterol it has very limited effect on the amount of cytotoxicity induced (Figure 4).
Bcl-2 Positive_regulation (induced) of Gene_expression (expression) of BetA associated with apoptosis
1) Confidence 0.04 Published 2009 Journal PLoS ONE Section Body Doc Link PMC2671171 Disease Relevance 0.63 Pain Relevance 0

General Comments

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