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Context Info
Confidence 0.16
First Reported 2008
Last Reported 2009
Negated 2
Speculated 1
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 1.46
Pain Relevance 0.57

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (AGTR1) signal transducer activity (AGTR1)
Anatomy Link Frequency
tail 2
Ankle 2
AGTR1 (Homo sapiens)
Pain Link Frequency Relevance Heat
antagonist 19 100.00 Very High Very High Very High
agonist 106 99.80 Very High Very High Very High
Onset of action 1 91.72 High High
headache 3 84.24 Quite High
Dopamine 1 69.92 Quite High
imagery 7 29.52 Quite Low
ischemia 2 5.00 Very Low Very Low Very Low
dopamine receptor 1 5.00 Very Low Very Low Very Low
cINOD 1 5.00 Very Low Very Low Very Low
bradykinin 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Pressure And Volume Under Development 8 99.90 Very High Very High Very High
Hypertension 43 99.16 Very High Very High Very High
Increased Venous Pressure Under Development 7 85.32 High High
Heart Rate Under Development 5 84.80 Quite High
Headache 3 84.24 Quite High
Cv Unclassified Under Development 6 38.28 Quite Low
Aging 1 19.84 Low Low
Stroke 4 5.00 Very Low Very Low Very Low
Myocardial Infarction 4 5.00 Very Low Very Low Very Low
Renal Artery Obstruction 3 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Likewise, agonist-mediated internalization of the angiotensin II type 1A receptor (AT1AR) was not affected by Rab5 protein expression despite the formation of Rab5-AT1AR protein complexes mediated, at least in part, by the last 10 amino acid residues of the C-tail of AT1AR itself [37].
Neg (not) Regulation (affected) of Localization (internalization) of AT1AR in tail associated with agonist
1) Confidence 0.16 Published 2008 Journal Biochim Biophys Acta Section Body Doc Link PMC2680976 Disease Relevance 0 Pain Relevance 0.12
Ankle edema encountered during calcium entry blockade is not due to renal fluid retention but involves a rise in intracapillary pressure (as a consequence of a selective diminution of the precapillary arteriolar tone), with an ensuing leak of fluid into the interstitium.22 Valsartan, like all ARBs, exhibits a placebo-like tolerability.8,17

Position of calcium antagonists and angiotensin receptor blockers in the treatment of hypertension

Neg (not) Spec (like) Regulation (involves) of Localization (blockers) of angiotensin receptor in Ankle associated with pressure and volume under development, antagonist and hypertension
2) Confidence 0.10 Published 2009 Journal Vascular Health and Risk Management Section Body Doc Link PMC2672450 Disease Relevance 1.46 Pain Relevance 0.45

General Comments

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