INT268126

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Context Info
Confidence 0.02
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 1.07
Pain Relevance 0.33

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell proliferation (OCA2) transport (OCA2) transmembrane transport (OCA2)
cytoplasm (OCA2)
Anatomy Link Frequency
neurons 1
OCA2 (Homo sapiens)
Pain Link Frequency Relevance Heat
Kinase C 130 100.00 Very High Very High Very High
GABAergic 2 53.16 Quite High
antagonist 4 50.52 Quite High
Central nervous system 20 5.00 Very Low Very Low Very Low
Inflammation 6 5.00 Very Low Very Low Very Low
Neuronal excitability 4 5.00 Very Low Very Low Very Low
Hippocampus 4 5.00 Very Low Very Low Very Low
imagery 4 5.00 Very Low Very Low Very Low
tetrodotoxin 2 5.00 Very Low Very Low Very Low
long-term potentiation 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Virus Diseases 178 100.00 Very High Very High Very High
Infection 58 92.96 High High
Borna Disease 8 50.00 Quite Low
Disease 12 5.00 Very Low Very Low Very Low
Neurological Disease 8 5.00 Very Low Very Low Very Low
Sprains And Strains 6 5.00 Very Low Very Low Very Low
Cognitive Disorder 6 5.00 Very Low Very Low Very Low
Viral Infection 6 5.00 Very Low Very Low Very Low
INFLAMMATION 6 5.00 Very Low Very Low Very Low
Congenital Anomalies 4 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Therefore, a recombinant BDV which can no longer be phosphorylated by PKC on its mutated P protein has lost its capacity to block activity-induced synaptic potentiation.


Phosphorylation (phosphorylated) of P protein associated with virus diseases and kinase c
1) Confidence 0.02 Published 2009 Journal PLoS Pathogens Section Body Doc Link PMC2673689 Disease Relevance 0.49 Pain Relevance 0.14
Using a recombinant virus in which the PKC phosphorylation sites of BDV P protein have been destroyed, we demonstrate that primary cultures of neurons infected with this recombinant virus exhibit a behavior that becomes indistinguishable in many aspects to that of control, non-infected neurons.
Phosphorylation (phosphorylation) of P protein in neurons associated with virus diseases and kinase c
2) Confidence 0.01 Published 2009 Journal PLoS Pathogens Section Body Doc Link PMC2673689 Disease Relevance 0.57 Pain Relevance 0.19

General Comments

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