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Context Info
Confidence 0.12
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 1.40
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (ARAF) protein modification process (ARAF) mitochondrion (ARAF)
cellular_component (ARAF)
ARAF (Homo sapiens)
Pain Link Frequency Relevance Heat
COX2 1 5.00 Very Low Very Low Very Low
abdominal pain 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Cancer 31 97.24 Very High Very High Very High
Chromosomal Instability 3 86.80 High High
Colorectal Cancer 50 84.44 Quite High
Hereditary Nonpolyposis Colorectal Neoplasms 11 75.64 Quite High
Familial Adenomatous Polyposis 48 74.88 Quite High
Syndrome 38 74.40 Quite High
Hantavirus Infection 7 67.36 Quite High
Polyps 35 50.76 Quite High
Hyperplasia 15 50.48 Quite High
Adenoma 33 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In their model, it was suggested that mutation of the k-ras proto-oncogene leading to activation of the oncogene as well as mutational inactivation of tumour suppressor genes existing on chromosomes 5q (APC), 7p (p53) and 18q (SMAD4) were the key initiators of colorectal carcinogenesis.
Positive_regulation (leading) of Gene_expression (mutation) of oncogene associated with cancer
1) Confidence 0.12 Published 2008 Journal Current Genomics Section Body Doc Link PMC2674304 Disease Relevance 1.40 Pain Relevance 0

General Comments

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