INT26948

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Context Info
Confidence 0.22
First Reported 1981
Last Reported 1989
Negated 0
Speculated 0
Reported most in Abstract
Documents 2
Total Number 2
Disease Relevance 0.15
Pain Relevance 1.99

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular space (Pomc, Gh1) signal transduction (Pomc, Gh1) extracellular region (Pomc, Gh1)
mitochondrion (Gh1) cell-cell signaling (Pomc) cytoplasm (Pomc)
Pomc (Rattus norvegicus)
Gh1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Opioid 7 100.00 Very High Very High Very High
Enkephalin 1 100.00 Very High Very High Very High
Clonidine 10 98.16 Very High Very High Very High
opioid receptor 1 95.12 Very High Very High Very High
anesthesia 1 94.64 High High
narcan 6 93.44 High High
opiate 1 92.40 High High
antagonist 2 91.12 High High
substance P 1 86.00 High High
Somatostatin 1 85.28 High High
Disease Link Frequency Relevance Heat
Body Weight 2 77.64 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Our data suggest that beta-endorphin or a related opioid peptide is an important mediator of GH secretion induced by alpha2-adrenergic stimulation.
beta-endorphin Positive_regulation (mediator) of Localization (secretion) of GH associated with opioid
1) Confidence 0.22 Published 1989 Journal Neuroendocrinology Section Abstract Doc Link 2572990 Disease Relevance 0.15 Pain Relevance 1.10
Both beta-endorphin and enkephalins are capable of stimulating GH and PRL release in anesthetized and unanesthetized, freely moving rats.
beta-endorphin Positive_regulation (stimulating) of Localization (release) of GH associated with enkephalin
2) Confidence 0.02 Published 1981 Journal Adv. Biochem. Psychopharmacol. Section Abstract Doc Link 7010947 Disease Relevance 0 Pain Relevance 0.88

General Comments

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