INT269496

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Context Info
Confidence 0.25
First Reported 2009
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 3
Total Number 3
Disease Relevance 0.46
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (Nos3) Golgi apparatus (Nos3) cytoplasm (Nos3)
signal transduction (Nos3) oxidoreductase activity (Nos3) nucleolus (Nos3)
Anatomy Link Frequency
cardiomyocyte 4
Nos3 (Mus musculus)
Pain Link Frequency Relevance Heat
cytokine 26 12.36 Low Low
cva 6 5.00 Very Low Very Low Very Low
anesthesia 5 5.00 Very Low Very Low Very Low
Inflammation 5 5.00 Very Low Very Low Very Low
ischemia 4 5.00 Very Low Very Low Very Low
cOX1 4 5.00 Very Low Very Low Very Low
fibrosis 3 5.00 Very Low Very Low Very Low
alcohol 3 5.00 Very Low Very Low Very Low
Neuronal nitric oxide synthase 2 5.00 Very Low Very Low Very Low
isoflurane 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Disease 15 93.92 High High
Pulmonary Disease 5 92.96 High High
Hypertension 27 72.72 Quite High
Adhesions 2 66.64 Quite High
Bacillus Anthracis Infection 28 50.00 Quite Low
Infection 8 42.88 Quite Low
Stress 11 20.08 Low Low
Shock 24 5.00 Very Low Very Low Very Low
Sprains And Strains 24 5.00 Very Low Very Low Very Low
Hypoxia 23 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Estrogen has been shown to influence the expression and activity of endothelial nitric oxide synthase (eNOS) which is associated with increased S-nitrosothiol production.
Regulation (influence) of eNOS Binding (associated) of
1) Confidence 0.25 Published 2010 Journal PLoS ONE Section Abstract Doc Link PMC2982841 Disease Relevance 0.33 Pain Relevance 0
In the heart eNOS is associated with the caveolae [29],[57] and nNOS is normally in the sarcoplasmic reticulum (SR) in close association with ryanodine receptors [29],[58] and XOR [37],[38]. nNOS-derived NO mediates cardiomyocyte contraction through its effects on the SR Ca+2 cycle that is central to excitation-contraction coupling and modulation of ryanodine receptor function [21], [30]–[32],[59].
Regulation (through) of eNOS Binding (associated) of in cardiomyocyte
2) Confidence 0.16 Published 2009 Journal PLoS Pathogens Section Body Doc Link PMC2680977 Disease Relevance 0.07 Pain Relevance 0
In the heart eNOS is associated with the caveolae [29],[57] and nNOS is normally in the sarcoplasmic reticulum (SR) in close association with ryanodine receptors [29],[58] and XOR [37],[38]. nNOS-derived NO mediates cardiomyocyte contraction through its effects on the SR Ca+2 cycle that is central to excitation-contraction coupling and modulation of ryanodine receptor function [21], [30]–[32],[59].
Regulation (effects) of eNOS Binding (associated) of in cardiomyocyte
3) Confidence 0.16 Published 2009 Journal PLoS Pathogens Section Body Doc Link PMC2680977 Disease Relevance 0.07 Pain Relevance 0

General Comments

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