INT269932

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Context Info
Confidence 0.09
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0.07
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleus (Pcna, Cdkn2b) cytoplasm (Pcna, Cdkn2b) intracellular (Pcna)
cell cycle (Cdkn2b) DNA binding (Pcna) kinase activity (Cdkn2b)
Pcna (Mus musculus)
Cdkn2b (Mus musculus)
Pain Link Frequency Relevance Heat
ketamine 2 5.00 Very Low Very Low Very Low
anesthesia 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apoptosis 2 67.96 Quite High
Injury 4 16.88 Low Low
Repression 14 5.00 Very Low Very Low Very Low
Targeted Disruption 8 5.00 Very Low Very Low Very Low
Hyperplasia 4 5.00 Very Low Very Low Very Low
Exsanguination 2 5.00 Very Low Very Low Very Low
Breast Cancer 2 5.00 Very Low Very Low Very Low
Cancer 2 5.00 Very Low Very Low Very Low
Hypoglycemia 2 5.00 Very Low Very Low Very Low
Colon Cancer 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
For example, Smad2, 3, and 4 directly activate the promoters of the cyclin-dependent kinase inhibitors p15 and p21, which in turn inhibit cdk2/4/6-cyclin complex activity [21]–[23].
cyclin Negative_regulation (inhibitors) of p15
1) Confidence 0.09 Published 2009 Journal PLoS ONE Section Body Doc Link PMC2682659 Disease Relevance 0.07 Pain Relevance 0
has been attributed, at least in part, to Smad-dependent transcriptional induction of cyclin-dependent kinase inhibitors including p15 and p21, leading to G1 arrest [21]–[23].
cyclin Negative_regulation (inhibitors) of p15
2) Confidence 0.09 Published 2009 Journal PLoS ONE Section Body Doc Link PMC2682659 Disease Relevance 0 Pain Relevance 0

General Comments

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