INT270619

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.32
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.89
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (Il21r)
Il21r (Mus musculus)
Pain Link Frequency Relevance Heat
chemokine 7 5.00 Very Low Very Low Very Low
Inflammation 5 5.00 Very Low Very Low Very Low
Inflammatory response 2 5.00 Very Low Very Low Very Low
Glutamate 2 5.00 Very Low Very Low Very Low
diabetic neuropathy 2 5.00 Very Low Very Low Very Low
member 8 1 5.00 Very Low Very Low Very Low
adenocard 1 5.00 Very Low Very Low Very Low
agonist 1 5.00 Very Low Very Low Very Low
Crohn's disease 1 5.00 Very Low Very Low Very Low
Arthritis 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Genetic Predisposition To Disease 4 99.76 Very High Very High Very High
Diabetes Mellitus 154 98.72 Very High Very High Very High
Sprains And Strains 4 96.64 Very High Very High Very High
Disease 8 95.96 Very High Very High Very High
Autoimmune Disease 15 7.60 Low Low
INFLAMMATION 7 5.00 Very Low Very Low Very Low
Diabetes Mellitus-type I 7 5.00 Very Low Very Low Very Low
Renal Disease 5 5.00 Very Low Very Low Very Low
Hyperglycemia 5 5.00 Very Low Very Low Very Low
Apoptosis 4 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Data suggest a contribution of IL21 and IL21R to a genetic susceptibility to T1D and possible involvement of IL-21 and its receptor system in the disease pathogenesis [42].
Positive_regulation (contribution) of IL21R associated with diabetes mellitus, genetic predisposition to disease and disease
1) Confidence 0.32 Published 2008 Journal Current Genomics Section Body Doc Link PMC2685644 Disease Relevance 0.89 Pain Relevance 0

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox