INT270731

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Context Info
Confidence 0.02
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.67
Pain Relevance 0.09

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (AGTR1, ACE) endosome (ACE) peptidase activity (ACE)
extracellular space (ACE) extracellular region (ACE) signal transducer activity (AGTR1)
AGTR1 (Homo sapiens)
ACE (Homo sapiens)
Pain Link Frequency Relevance Heat
fibrosis 1 94.24 High High
Inflammation 4 91.04 High High
beta blocker 5 5.00 Very Low Very Low Very Low
Angina 3 5.00 Very Low Very Low Very Low
Calcium channel 3 5.00 Very Low Very Low Very Low
ischemia 2 5.00 Very Low Very Low Very Low
withdrawal 1 5.00 Very Low Very Low Very Low
Inflammatory marker 1 5.00 Very Low Very Low Very Low
Bioavailability 1 5.00 Very Low Very Low Very Low
tolerance 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Internal Fibrosis 1 94.24 High High
Vasculitis 1 91.04 High High
Heart Rate Under Development 4 87.28 High High
Increased Venous Pressure Under Development 11 85.40 High High
Chronic Renal Failure 3 84.24 Quite High
Diabetes Mellitus 110 82.32 Quite High
Natriuresis 2 73.04 Quite High
Ventricular Remodeling 1 68.16 Quite High
Hypertension 68 50.00 Quite Low
Proteinuria 12 48.12 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Although ACE inhibitors are able to reduce angiotensin II formation, non-ACE dependent pathways have also been identified.137 On the other hand, ARBs antagonize the binding of angiotensin II to the AT1 receptor, which mediates most of the undesirable effects associated with angiotensin II.
AT1 receptor Binding (antagonize) of non-ACE
1) Confidence 0.02 Published 2009 Journal Vascular Health and Risk Management Section Body Doc Link PMC2686259 Disease Relevance 0.67 Pain Relevance 0.09

General Comments

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