INT270763

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Context Info
Confidence 0.04
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 1.13
Pain Relevance 0.66

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (IL1B, IKBKB) aging (IL1B) cytoplasm (IKBKB)
cell-cell signaling (IL1B) extracellular space (IL1B) signal transduction (IL1B)
IL1B (Homo sapiens)
IKBKB (Homo sapiens)
Pain Link Frequency Relevance Heat
Inflammation 6 99.68 Very High Very High Very High
Pain 76 94.40 High High
Osteoarthritis 106 91.16 High High
Inflammatory marker 2 88.24 High High
Arthritis 10 18.24 Low Low
Paracetamol 16 5.00 Very Low Very Low Very Low
cINOD 12 5.00 Very Low Very Low Very Low
withdrawal 4 5.00 Very Low Very Low Very Low
Pain score 4 5.00 Very Low Very Low Very Low
Analgesic 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
INFLAMMATION 10 99.68 Very High Very High Very High
Pain 78 94.40 High High
Osteoarthritis 112 91.16 High High
Arthritis 10 18.24 Low Low
Disease 36 5.00 Very Low Very Low Very Low
Pressure And Volume Under Development 8 5.00 Very Low Very Low Very Low
Obesity 6 5.00 Very Low Very Low Very Low
Aging 4 5.00 Very Low Very Low Very Low
Vomiting 4 5.00 Very Low Very Low Very Low
Toxicity 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
More recently, however, it has been demonstrated in animal models that glucosamine has an anti-inflammatory effect via the reduction of nuclear factor kappa beta induced by interleukin-1 (IL-1) (Gouze et al 2002, 2006).
interleukin-1 Negative_regulation (reduction) of nuclear factor kappa beta associated with inflammation
1) Confidence 0.04 Published 2007 Journal Clinical Interventions in Aging Section Body Doc Link PMC2686334 Disease Relevance 0.56 Pain Relevance 0.33
More recently, however, it has been demonstrated in animal models that glucosamine has an anti-inflammatory effect via the reduction of nuclear factor kappa beta induced by interleukin-1 (IL-1) (Gouze et al 2002, 2006).
IL-1 Negative_regulation (reduction) of nuclear factor kappa beta associated with inflammation
2) Confidence 0.04 Published 2007 Journal Clinical Interventions in Aging Section Body Doc Link PMC2686334 Disease Relevance 0.57 Pain Relevance 0.33

General Comments

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