INT272750

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Context Info
Confidence 0.02
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0.11

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleus (Prkaca, EGFR) plasma membrane (Prkaca, EGFR) Golgi apparatus (Prkaca, EGFR)
cytoplasm (Prkaca, EGFR) mitochondrion (Prkaca) extracellular space (EGFR)
Prkaca (Mus musculus)
EGFR (Homo sapiens)
Pain Link Frequency Relevance Heat
antagonist 50 58.32 Quite High
Inflammation 3 5.00 Very Low Very Low Very Low
withdrawal 2 5.00 Very Low Very Low Very Low
agonist 2 5.00 Very Low Very Low Very Low
cerebral cortex 1 5.00 Very Low Very Low Very Low
adenocard 1 5.00 Very Low Very Low Very Low
Paracetamol 1 5.00 Very Low Very Low Very Low
Kinase C 1 5.00 Very Low Very Low Very Low
imagery 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Cancer 7 36.60 Quite Low
Adenocarcinoma 9 11.64 Low Low
Endometriosis (extended) 8 7.20 Low Low
INFLAMMATION 3 5.00 Very Low Very Low Very Low
Reprotox - General 1 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
stimulation of FPS cells induces ERK1/2 phosphorylation via identical intracellular signaling pathways via the PLC-PKA-mediated activation of the EGFR since co-treatment of cells with the PLC, PKA or EGFR kinase inhibitors significantly inhibited the PGE2 or PGF2?
PLC-PKA-mediated Positive_regulation (activation) of EGFR
1) Confidence 0.02 Published 2008 Journal Molecular and Cellular Endocrinology Section Body Doc Link PMC2694994 Disease Relevance 0 Pain Relevance 0.11

General Comments

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