INT273698

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Context Info
Confidence 0.39
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0.22
Pain Relevance 0.08

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell adhesion (Itgb2)
Anatomy Link Frequency
proximal 2
vesicle 2
Itgb2 (Mus musculus)
Pain Link Frequency Relevance Heat
antagonist 14 91.68 High High
Inflammation 8 45.00 Quite Low
antidepressant 12 38.00 Quite Low
Kinase C 2 20.32 Low Low
Serotonin 4 13.12 Low Low
sSRI 2 10.00 Low Low
fluoxetine 4 5.00 Very Low Very Low Very Low
Desipramine 2 5.00 Very Low Very Low Very Low
imagery 2 5.00 Very Low Very Low Very Low
Potency 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Targeted Disruption 12 76.60 Quite High
Adhesions 4 66.04 Quite High
Disease 2 49.32 Quite Low
Autoimmune Disease 6 45.96 Quite Low
INFLAMMATION 8 45.00 Quite Low
Immune System Diseases 2 43.84 Quite Low
Infection 2 32.08 Quite Low
Syndrome 4 5.00 Very Low Very Low Very Low
Hypersensitivity 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The pilot scale HTS validated the usefulness of our HTS platform in the systematic investigation of molecular mechanisms controlling TCR-mediated membrane-proximal signaling cascades leading to LFA-1 activation.
Positive_regulation (leading) of Positive_regulation (activation) of LFA-1 in proximal
1) Confidence 0.39 Published 2009 Journal PLoS ONE Section Body Doc Link PMC2698288 Disease Relevance 0 Pain Relevance 0.08
Further, in this study, we reveal that the vesicle-absorption also involves intracellular signaling cascades initiated by TCR triggering and required for LFA-1 activation.
Positive_regulation (required) of Positive_regulation (activation) of LFA-1 in vesicle
2) Confidence 0.26 Published 2009 Journal PLoS ONE Section Body Doc Link PMC2698288 Disease Relevance 0.22 Pain Relevance 0

General Comments

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