INT274216

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Context Info
Confidence 0.08
First Reported 2008
Last Reported 2008
Negated 1
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.27
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (Gsr) mitochondrion (Gsr) signal transduction (Mc3r)
oxidoreductase activity (Gsr) plasma membrane (Mc3r) cytoplasm (Gsr)
Anatomy Link Frequency
granulocytes 2
Mc3r (Mus musculus)
Gsr (Mus musculus)
Pain Link Frequency Relevance Heat
ischemia 6 50.00 Quite Low
chemokine 11 48.92 Quite Low
Inflammation 49 8.24 Low Low
agonist 14 5.00 Very Low Very Low Very Low
cytokine 6 5.00 Very Low Very Low Very Low
Inflammatory response 6 5.00 Very Low Very Low Very Low
adenocard 2 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
alcohol 1 5.00 Very Low Very Low Very Low
Inflammatory stimuli 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Injury 7 100.00 Very High Very High Very High
Adhesions 15 92.72 High High
Cv Unclassified Under Development 6 50.00 Quite Low
INFLAMMATION 53 8.24 Low Low
Urological Neuroanatomy 4 5.00 Very Low Very Low Very Low
Vasculitis 3 5.00 Very Low Very Low Very Low
Sprains And Strains 2 5.00 Very Low Very Low Very Low
Arthritis 1 5.00 Very Low Very Low Very Low
Necrosis 1 5.00 Very Low Very Low Very Low
Peripheral Arterial Disease 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Within the GR-1hi population, CD11b expression was not modified by the IR injury in either WT or MC3R-null animals; however, granulocytes from IR MC3R-null animals displayed an increased CD11b expression when compared with WT animals.
MC3R-null Neg (not) Regulation (modified) of Gene_expression (expression) of GR-1hi in granulocytes associated with injury
1) Confidence 0.08 Published 2008 Journal FASEB J Section Body Doc Link PMC2700033 Disease Relevance 0.27 Pain Relevance 0

General Comments

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