INT27543

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Context Info
Confidence 0.09
First Reported 1989
Last Reported 1989
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 2
Disease Relevance 2.09
Pain Relevance 0.48

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (ALOX12) oxidoreductase activity (ALOX12) cytoplasm (ALOX12)
Anatomy Link Frequency
platelet 1
heart 1
ALOX12 (Homo sapiens)
Pain Link Frequency Relevance Heat
Angina 6 82.52 Quite High
Disease Link Frequency Relevance Heat
Coronary Artery Disease 12 99.46 Very High Very High Very High
Syndrome 8 98.00 Very High Very High Very High
Stable Angina Pectoris 2 82.52 Quite High
Cv General 3 Under Development 4 81.04 Quite High
Infarction 2 78.28 Quite High
Adhesions 2 75.00 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The abnormal increase of platelet GPlb is due to pathological mechanism of blood stasis syndrome of coronary heart disease.
Positive_regulation (increase) of platelet GPlb in heart associated with coronary artery disease and syndrome
1) Confidence 0.09 Published 1989 Journal Zhong Xi Yi Jie He Za Zhi Section Abstract Doc Link 2624983 Disease Relevance 1.13 Pain Relevance 0.23
These results suggested that it played an important part in the occurrence and development of coronary heart disease as manifested by abnormal increase of platelet GPlb, and enhancement of platelet's adherence and aggregation.
Positive_regulation (increase) of platelet GPlb in platelet associated with coronary artery disease
2) Confidence 0.06 Published 1989 Journal Zhong Xi Yi Jie He Za Zhi Section Abstract Doc Link 2624983 Disease Relevance 0.97 Pain Relevance 0.24

General Comments

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