INT276668

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Context Info
Confidence 0.09
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.98
Pain Relevance 1.85

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (Ngf) extracellular space (Ngf) aging (Ngf)
extracellular region (Ngf) molecular_function (Ngf)
Ngf (Rattus norvegicus)
Nav1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
nav1.8 65 100.00 Very High Very High Very High
Inflammatory mediators 1 100.00 Very High Very High Very High
Serotonin 1 100.00 Very High Very High Very High
Kinase C 38 99.08 Very High Very High Very High
Pain 11 97.48 Very High Very High Very High
Action potential 7 91.36 High High
agonist 4 90.64 High High
dorsal root ganglion 43 88.32 High High
sodium channel 5 86.60 High High
tetrodotoxin 17 85.60 High High
Disease Link Frequency Relevance Heat
INFLAMMATION 11 100.00 Very High Very High Very High
Pain 13 97.48 Very High Very High Very High
Ganglion Cysts 45 88.32 High High
Nervous System Injury 1 57.52 Quite High
Nociception 3 46.96 Quite Low
Hyperalgesia 5 34.84 Quite Low
Rupture 1 6.00 Low Low
Neuropathic Pain 3 5.00 Very Low Very Low Very Low
Inflammatory Pain 3 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Accumulative evidence showed that the Nav1.8 current was regulated by various inflammatory mediators, such as prostaglandin E2 (PGE2), serotonin, NGF etc. through a PKA or PKC signaling pathway [21-24].
NGF Regulation (regulated) of Nav1.8 associated with kinase c, inflammatory mediators, nav1.8 and serotonin
1) Confidence 0.09 Published 2009 Journal Mol Pain Section Body Doc Link PMC2715383 Disease Relevance 0.98 Pain Relevance 1.85

General Comments

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