INT276672

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Context Info
Confidence 0.16
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.18
Pain Relevance 1.67

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (Prkcg) plasma membrane (Prkcg) nucleus (Prkcg)
intracellular (Prkcg) response to stress (Prkcg) cytoplasm (Prkcg)
Prkcg (Rattus norvegicus)
Nav1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
nav1.8 65 100.00 Very High Very High Very High
Kinase C 38 100.00 Very High Very High Very High
substance P 66 98.96 Very High Very High Very High
dorsal root ganglion 43 94.48 High High
addiction 3 70.20 Quite High
Lasting pain 2 32.16 Quite Low
Inflammation 10 31.64 Quite Low
tetrodotoxin 17 5.00 Very Low Very Low Very Low
Pain 11 5.00 Very Low Very Low Very Low
Action potential 7 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Ganglion Cysts 45 94.48 High High
Pain 13 32.16 Quite Low
INFLAMMATION 11 31.64 Quite Low
Hyperalgesia 5 5.00 Very Low Very Low Very Low
Nociception 3 5.00 Very Low Very Low Very Low
Neuropathic Pain 3 5.00 Very Low Very Low Very Low
Inflammatory Pain 3 5.00 Very Low Very Low Very Low
Nervous System Injury 1 5.00 Very Low Very Low Very Low
Rupture 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
M) for 30 min before Sar-SP perfusion, Sar-SP failed to potentiate Nav1.8 currents, suggesting a PKC-dependent mechanism in the interaction of NK-1 and Nav1.8 (Figure 3A and 3B).
PKC Binding (interaction) of Nav1.8 associated with kinase c, nav1.8 and substance p
1) Confidence 0.16 Published 2009 Journal Mol Pain Section Body Doc Link PMC2715383 Disease Relevance 0.18 Pain Relevance 1.67

General Comments

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