INT276673

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.05
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.16
Pain Relevance 1.37

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (Trpv1) plasma membrane (Trpv1) nucleus (Nkx1-1)
neurological system process (Nkx1-1) lipid metabolic process (Trpv1)
Nkx1-1 (Rattus norvegicus)
Trpv1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Kinase C 38 97.64 Very High Very High Very High
nav1.8 65 93.32 High High
Pain 11 87.72 High High
Peripheral sensitization 1 87.40 High High
substance P 66 79.92 Quite High
calcitonin gene related peptide 1 78.96 Quite High
Neuropeptide 2 77.68 Quite High
hyperexcitability 1 59.96 Quite High
Action potential 7 57.88 Quite High
tetrodotoxin 17 45.12 Quite Low
Disease Link Frequency Relevance Heat
Pain 13 87.72 High High
Ganglion Cysts 45 50.00 Quite Low
Inflammatory Pain 3 43.60 Quite Low
Neuropathic Pain 3 42.40 Quite Low
Hyperalgesia 5 41.80 Quite Low
INFLAMMATION 11 36.64 Quite Low
Nociception 3 28.28 Quite Low
Nervous System Injury 1 5.00 Very Low Very Low Very Low
Rupture 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In agreement with our previous finding that the modulation of TRPV1 by NK-1 receptor was mediated by activation of PLC and downstream PKC pathway [9], the present results also proved the involvement of PKC in the interaction between NK-1 and Nav1.8.
NK-1 Regulation (modulation) of TRPV1 associated with kinase c and nav1.8
1) Confidence 0.05 Published 2009 Journal Mol Pain Section Body Doc Link PMC2715383 Disease Relevance 0.16 Pain Relevance 1.37

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox