INT276675

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Context Info
Confidence 0.09
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.21
Pain Relevance 1.46

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleus (Nkx1-1) neurological system process (Nkx1-1)
Nkx1-1 (Rattus norvegicus)
Nav1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
nav1.8 65 100.00 Very High Very High Very High
Kinase C 38 99.84 Very High Very High Very High
substance P 66 93.76 High High
Pain 11 81.12 Quite High
Peripheral sensitization 1 80.80 Quite High
calcitonin gene related peptide 1 72.36 Quite High
Neuropeptide 2 71.08 Quite High
tetrodotoxin 17 58.96 Quite High
dorsal root ganglion 43 56.64 Quite High
hyperexcitability 1 53.36 Quite High
Disease Link Frequency Relevance Heat
Pain 13 81.12 Quite High
Ganglion Cysts 45 56.64 Quite High
Inflammatory Pain 3 37.00 Quite Low
Neuropathic Pain 3 35.80 Quite Low
Hyperalgesia 5 35.20 Quite Low
INFLAMMATION 11 30.04 Quite Low
Nociception 3 21.68 Low Low
Nervous System Injury 1 5.00 Very Low Very Low Very Low
Rupture 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In agreement with our previous finding that the modulation of TRPV1 by NK-1 receptor was mediated by activation of PLC and downstream PKC pathway [9], the present results also proved the involvement of PKC in the interaction between NK-1 and Nav1.8.
NK-1 Binding (interaction) of Nav1.8 associated with kinase c and nav1.8
1) Confidence 0.09 Published 2009 Journal Mol Pain Section Body Doc Link PMC2715383 Disease Relevance 0.21 Pain Relevance 1.46

General Comments

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