INT276679

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Context Info
Confidence 0.07
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0.94
Pain Relevance 3.08

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleus (Prkcg, Nkx1-1) cytosol (Prkcg) plasma membrane (Prkcg)
intracellular (Prkcg) neurological system process (Nkx1-1) response to stress (Prkcg)
Prkcg (Rattus norvegicus)
Nkx1-1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Kinase C 76 100.00 Very High Very High Very High
nav1.8 130 97.28 Very High Very High Very High
dorsal root ganglion 86 93.84 High High
substance P 132 92.32 High High
IPN 6 92.00 High High
tetrodotoxin 34 87.44 High High
Inflammation 20 78.48 Quite High
Pain 22 38.48 Quite Low
Spinal cord 8 36.08 Quite Low
sodium channel 10 27.36 Quite Low
Disease Link Frequency Relevance Heat
Ganglion Cysts 90 93.84 High High
Inflammatory Pain 6 92.00 High High
INFLAMMATION 22 78.48 Quite High
Nociception 6 43.28 Quite Low
Pain 26 38.48 Quite Low
Hyperalgesia 10 5.00 Very Low Very Low Very Low
Neuropathic Pain 6 5.00 Very Low Very Low Very Low
Nervous System Injury 2 5.00 Very Low Very Low Very Low
Rupture 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Therefore, the modulatory action of NK-1 may be predominately mediated by PKC, particularly by PKC?
PKC Positive_regulation (mediated) of Positive_regulation (action) of NK-1 associated with kinase c
1) Confidence 0.07 Published 2009 Journal Mol Pain Section Body Doc Link PMC2715383 Disease Relevance 0.47 Pain Relevance 1.53
Therefore, the modulatory action of NK-1 may be predominately mediated by PKC, particularly by PKC?
PKC Positive_regulation (mediated) of Positive_regulation (action) of NK-1 associated with kinase c
2) Confidence 0.07 Published 2009 Journal Mol Pain Section Body Doc Link PMC2715383 Disease Relevance 0.47 Pain Relevance 1.54

General Comments

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