INT276682

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Context Info
Confidence 0.11
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 1.07
Pain Relevance 2.00

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleus (Nkx1-1) neurological system process (Nkx1-1)
Nkx1-1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
nav1.8 65 100.00 Very High Very High Very High
IPN 3 97.28 Very High Very High Very High
Hyperalgesia 5 95.48 Very High Very High Very High
allodynia 1 94.80 High High
conotoxin 1 92.56 High High
Neuropathic pain 1 91.16 High High
Inflammation 10 90.32 High High
Action potential 7 82.56 Quite High
hyperexcitability 1 78.48 Quite High
Pain 11 76.32 Quite High
Disease Link Frequency Relevance Heat
Inflammatory Pain 3 97.28 Very High Very High Very High
Neuropathic Pain 3 96.08 Very High Very High Very High
Hyperalgesia 5 95.48 Very High Very High Very High
INFLAMMATION 11 90.32 High High
Nociception 3 81.96 Quite High
Pain 13 76.32 Quite High
Ganglion Cysts 45 63.36 Quite High
Nervous System Injury 1 5.00 Very Low Very Low Very Low
Rupture 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The present study for the first time revealed that NK-1 activation potentiated Nav1.8 currents and shifted both the activation and steady-state inactivation curves of this channel in a hyperpolarizing direction.
Negative_regulation (shifted) of Positive_regulation (activation) of NK-1 associated with nav1.8
1) Confidence 0.11 Published 2009 Journal Mol Pain Section Body Doc Link PMC2715383 Disease Relevance 1.07 Pain Relevance 2.00

General Comments

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