INT276827

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Context Info
Confidence 0.24
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.65
Pain Relevance 0.95

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (Gpr65) plasma membrane (Gpr65) signal transducer activity (Gpr65)
Anatomy Link Frequency
neurons 1
Gpr65 (Mus musculus)
Pain Link Frequency Relevance Heat
IPN 28 99.04 Very High Very High Very High
Kinase C 9 94.80 High High
Inflammation 50 85.68 High High
qutenza 44 71.96 Quite High
Hyperalgesia 23 69.44 Quite High
ASIC 2 7.52 Low Low
Pain 14 5.00 Very Low Very Low Very Low
withdrawal 10 5.00 Very Low Very Low Very Low
nociceptor 7 5.00 Very Low Very Low Very Low
Thermal hyperalgesia 4 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Inflammatory Pain 28 99.04 Very High Very High Very High
INFLAMMATION 51 85.68 High High
Hyperalgesia 27 69.44 Quite High
Pressure And Volume Under Development 21 5.00 Very Low Very Low Very Low
Pain 16 5.00 Very Low Very Low Very Low
Nociception 9 5.00 Very Low Very Low Very Low
Hypersensitivity 6 5.00 Very Low Very Low Very Low
Bordatella Infection 2 5.00 Very Low Very Low Very Low
Acidosis 2 5.00 Very Low Very Low Very Low
Death 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Therefore, TDAG8 could be involved in sensitization of TRPV1 function in IB4-positive neurons and in the development of the chronic inflammatory pain state through the cAMP-PKC?
Regulation (involved) of TDAG8 in neurons associated with kinase c and ipn
1) Confidence 0.24 Published 2009 Journal Mol Pain Section Body Doc Link PMC2716309 Disease Relevance 0.65 Pain Relevance 0.95

General Comments

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