INT278573

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Context Info
Confidence 0.17
First Reported 2009
Last Reported 2009
Negated 0
Speculated 1
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0.07
Pain Relevance 0.09

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

endosome (Atp6v1e1) transport (Atp6v1e1) mitochondrion (Atp6v1e1)
signal transducer activity (Tacr3) cytoplasm (Atp6v1e1) cytosol (Atp6v1e1)
Atp6v1e1 (Mus musculus)
Tacr3 (Mus musculus)
Pain Link Frequency Relevance Heat
agonist 20 92.08 High High
substance P 22 41.56 Quite Low
Central nervous system 4 41.40 Quite Low
anesthesia 2 15.36 Low Low
Inflammatory mediators 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Hypogonadotropic Hypogonadism 2 71.36 Quite High
Reprotox - General 1 2 36.44 Quite Low
Endometriosis (extended) 2 19.12 Low Low
Increased Venous Pressure Under Development 2 5.00 Very Low Very Low Very Low
Eclampsia 2 5.00 Very Low Very Low Very Low
INFLAMMATION 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The observation that E2 caused a rapid and concomitant down-regulation of Tac2 and Tacr3 expression supports the existence of an important link between E2 and the NK3R/NKB activation pathway [38].
E2 Regulation (regulation) of Gene_expression (expression) of Tacr3
1) Confidence 0.17 Published 2009 Journal Reprod Biol Endocrinol Section Body Doc Link PMC2724541 Disease Relevance 0.07 Pain Relevance 0
In this study, we have analyzed the direct effects of the ovarian steroids E2 and P4 on the expression of Tac1, Tac2, Tac4, Tacr1, Tacr2 and Tacr3 in uteri from ovariectomized mice.
E2 Spec (analyzed) Regulation (effects) of Spec (analyzed) Gene_expression (expression) of Tacr3
2) Confidence 0.09 Published 2009 Journal Reprod Biol Endocrinol Section Body Doc Link PMC2724541 Disease Relevance 0 Pain Relevance 0.09

General Comments

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