INT278772

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Context Info
Confidence 0.42
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 5
Disease Relevance 0
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleus (NUP98, HOXA9) nucleoplasm (NUP98) nuclear envelope (NUP98)
small molecule metabolic process (NUP98) carbohydrate metabolic process (NUP98) cytoplasm (HOXA9)
NUP98 (Homo sapiens)
HOXA9 (Homo sapiens)
HOXA9 - N51S (2)
Pain Link Frequency Relevance Heat
cytokine 10 20.32 Low Low
cINOD 15 5.00 Very Low Very Low Very Low
aspirin 10 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Myeloid Leukemia 30 17.68 Low Low
Chronic Lymphoid Leukemia 10 15.80 Low Low
Hematologic Neoplasms 10 14.24 Low Low
Leukemia 40 5.00 Very Low Very Low Very Low
Cancer 35 5.00 Very Low Very Low Very Low
Cytomegalovirus Infection 15 5.00 Very Low Very Low Very Low
Lymphatic System Cancer 10 5.00 Very Low Very Low Very Low
Repression 5 5.00 Very Low Very Low Very Low
Adhesions 5 5.00 Very Low Very Low Very Low
INFLAMMATION 5 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Homeodomain-independent gene regulation by NUP98-HOXA9 in primary human CD34+ cells
NUP98 Regulation (regulation) of HOXA9
1) Confidence 0.42 Published 2009 Journal PLoS ONE Section Body Doc Link PMC2725295 Disease Relevance 0 Pain Relevance 0
Based on gene expression analysis, a group of 46 probe sets were identified that show similar dysregulation by NUP98-HOXA9 and NUP98-HOXA9/N51S (Fig. 11A and Tables S2–S5) and therefore presumably do not require direct DNA binding by the homeodomain for their dysregulation.
NUP98 Regulation (dysregulation) of HOXA9
2) Confidence 0.25 Published 2009 Journal PLoS ONE Section Body Doc Link PMC2725295 Disease Relevance 0 Pain Relevance 0
Based on gene expression analysis, a group of 46 probe sets were identified that show similar dysregulation by NUP98-HOXA9 and NUP98-HOXA9/N51S (Fig. 11A and Tables S2–S5) and therefore presumably do not require direct DNA binding by the homeodomain for their dysregulation.
NUP98 Regulation (dysregulation) of HOXA9
3) Confidence 0.25 Published 2009 Journal PLoS ONE Section Body Doc Link PMC2725295 Disease Relevance 0 Pain Relevance 0
Based on gene expression analysis, a group of 46 probe sets were identified that show similar dysregulation by NUP98-HOXA9 and NUP98-HOXA9/N51S (Fig. 11A and Tables S2–S5) and therefore presumably do not require direct DNA binding by the homeodomain for their dysregulation.
NUP98 Regulation (dysregulation) of HOXA9 (N51S)
4) Confidence 0.11 Published 2009 Journal PLoS ONE Section Body Doc Link PMC2725295 Disease Relevance 0 Pain Relevance 0
Based on gene expression analysis, a group of 46 probe sets were identified that show similar dysregulation by NUP98-HOXA9 and NUP98-HOXA9/N51S (Fig. 11A and Tables S2–S5) and therefore presumably do not require direct DNA binding by the homeodomain for their dysregulation.
NUP98 Regulation (dysregulation) of HOXA9 (N51S)
5) Confidence 0.11 Published 2009 Journal PLoS ONE Section Body Doc Link PMC2725295 Disease Relevance 0 Pain Relevance 0

General Comments

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