INT278807

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Context Info
Confidence 0.59
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleus (HOXA9) cytoplasm (HOXA9)
HOXA9 (Homo sapiens)
Pain Link Frequency Relevance Heat
cINOD 3 5.00 Very Low Very Low Very Low
aspirin 2 5.00 Very Low Very Low Very Low
cytokine 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Leukemia 8 48.20 Quite Low
Cancer 7 5.00 Very Low Very Low Very Low
Myeloid Leukemia 6 5.00 Very Low Very Low Very Low
Cytomegalovirus Infection 3 5.00 Very Low Very Low Very Low
Lymphatic System Cancer 2 5.00 Very Low Very Low Very Low
Hematologic Neoplasms 2 5.00 Very Low Very Low Very Low
Chronic Lymphoid Leukemia 2 5.00 Very Low Very Low Very Low
Repression 1 5.00 Very Low Very Low Very Low
Adhesions 1 5.00 Very Low Very Low Very Low
INFLAMMATION 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Taken together, these results suggest that the effects of NUP98-HOXA9 on gene transcription and cell transformation are mediated by at least two distinct mechanisms: one that involves promoter binding through the homeodomain with direct transcriptional activation, and another that depends predominantly on the NUP98 moiety and does not involve direct DNA binding.



Transcription (transcription) of HOXA9
1) Confidence 0.59 Published 2009 Journal PLoS ONE Section Abstract Doc Link PMC2725295 Disease Relevance 0 Pain Relevance 0

General Comments

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