INT279828

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Context Info
Confidence 0.39
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 1.64
Pain Relevance 0.26

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transport (Scn1a, Scn9a) plasma membrane (Scn1a) transmembrane transport (Scn9a)
Scn1a (Mus musculus)
Scn9a (Mus musculus)
Pain Link Frequency Relevance Heat
Nav1.7 10 88.68 High High
sodium channel 4 66.56 Quite High
Pain 9 5.00 Very Low Very Low Very Low
Paroxysmal extreme pain disorder 7 5.00 Very Low Very Low Very Low
primary erythermalgia 5 5.00 Very Low Very Low Very Low
anticonvulsant 3 5.00 Very Low Very Low Very Low
Migraine 2 5.00 Very Low Very Low Very Low
Central nervous system 2 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
fibrosis 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Febrile Convulsions 60 100.00 Very High Very High Very High
Epilepsy 22 100.00 Very High Very High Very High
Syndrome 46 92.92 High High
Myoclonic Epilepsies 2 80.00 Quite High
Disease 15 68.04 Quite High
Convulsion 72 62.96 Quite High
Targeted Disruption 2 48.40 Quite Low
Pain 9 5.00 Very Low Very Low Very Low
Partial Seizures 7 5.00 Very Low Very Low Very Low
Somatoform Disorder 7 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
This study provides evidence for a role of SCN9A in human epilepsies, both as a cause of FS and as a partner with SCN1A mutations.


SCN1A Binding (partner) of SCN9A associated with epilepsy and febrile convulsions
1) Confidence 0.39 Published 2009 Journal PLoS Genetics Section Abstract Doc Link PMC2730533 Disease Relevance 1.64 Pain Relevance 0.26

General Comments

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