INT279845

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Context Info
Confidence 0.39
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0.28
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleus (Nipbl) cell cycle (Nipbl)
Anatomy Link Frequency
embryonic stage 1
Nipbl (Mus musculus)
Pain Link Frequency Relevance Heat
medulla 10 25.72 Quite Low
Inflammation 6 5.00 Very Low Very Low Very Low
cytokine 2 5.00 Very Low Very Low Very Low
agonist 2 5.00 Very Low Very Low Very Low
Immobilon 2 5.00 Very Low Very Low Very Low
Pain 2 5.00 Very Low Very Low Very Low
alcohol 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Congenital Anomalies 46 99.40 Very High Very High Very High
Syndrome 140 94.60 High High
Obesity 10 5.00 Very Low Very Low Very Low
Convulsion 8 5.00 Very Low Very Low Very Low
Disease 8 5.00 Very Low Very Low Very Low
Ventricular Heart Septal Defects 6 5.00 Very Low Very Low Very Low
Heart Defects 6 5.00 Very Low Very Low Very Low
INFLAMMATION 6 5.00 Very Low Very Low Very Low
Disorders Of The Lacrimal System 4 5.00 Very Low Very Low Very Low
Craniofacial Abnormalities 4 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
As shown in Figure 8B, the data support the microarray results from the earlier embryonic stage, and indicate that most transcriptional changes in Nipbl+/?
Regulation (changes) of Nipbl in embryonic stage
1) Confidence 0.39 Published 2009 Journal PLoS Genetics Section Body Doc Link PMC2730539 Disease Relevance 0 Pain Relevance 0
Overall the data from multiple species strongly argue that pervasive developmental abnormalities result from remarkably small changes in NIPBL levels.


Regulation (changes) of NIPBL associated with congenital anomalies
2) Confidence 0.24 Published 2009 Journal PLoS Genetics Section Body Doc Link PMC2730539 Disease Relevance 0.28 Pain Relevance 0

General Comments

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