INT279871

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Context Info
Confidence 0.43
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 2
Disease Relevance 1.22
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleus (Nipbl) cell cycle (Nipbl)
Anatomy Link Frequency
liver 2
Nipbl (Mus musculus)
Pain Link Frequency Relevance Heat
medulla 10 5.00 Very Low Very Low Very Low
Inflammation 6 5.00 Very Low Very Low Very Low
cytokine 2 5.00 Very Low Very Low Very Low
agonist 2 5.00 Very Low Very Low Very Low
Immobilon 2 5.00 Very Low Very Low Very Low
Pain 2 5.00 Very Low Very Low Very Low
alcohol 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Congenital Anomalies 46 93.48 High High
Heart Defects 6 92.56 High High
Syndrome 140 89.32 High High
Ventricular Heart Septal Defects 6 49.36 Quite Low
Disease 8 45.76 Quite Low
Cognitive Disorder 2 42.72 Quite Low
Corneal Opacity 2 38.32 Quite Low
Keloid Scars 2 34.00 Quite Low
Obesity 10 25.00 Low Low
Craniofacial Abnormalities 4 25.00 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
These phenotypes remained stable through many generations of outcrossing, and occurred in the context of modest (25–35%) reductions in levels of Nipbl mRNA in every tissue measured (embryonic brain, MEFs, adult liver).
Negative_regulation (reductions) of Transcription (levels) of Nipbl in liver
1) Confidence 0.43 Published 2009 Journal PLoS Genetics Section Body Doc Link PMC2730539 Disease Relevance 0.60 Pain Relevance 0
These phenotypes arose despite a decrease in Nipbl transcript levels of only ?
Negative_regulation (decrease) of Transcription (levels) of Nipbl
2) Confidence 0.38 Published 2009 Journal PLoS Genetics Section Abstract Doc Link PMC2730539 Disease Relevance 0.61 Pain Relevance 0

General Comments

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