INT280834

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Context Info
Confidence 0.05
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.23
Pain Relevance 0.06

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

DNA binding (Jun) signal transducer activity (Avp) cytosol (Jun)
extracellular space (Avp) signal transduction (Avp) extracellular region (Avp)
Jun (Mus musculus)
Avp (Mus musculus)
Pain Link Frequency Relevance Heat
Kinase C 12 64.72 Quite High
cytokine 13 5.00 Very Low Very Low Very Low
Neuropeptide 9 5.00 Very Low Very Low Very Low
dexamethasone 9 5.00 Very Low Very Low Very Low
amygdala 8 5.00 Very Low Very Low Very Low
medulla 4 5.00 Very Low Very Low Very Low
cerebral cortex 3 5.00 Very Low Very Low Very Low
Inflammation 3 5.00 Very Low Very Low Very Low
Spinal cord 2 5.00 Very Low Very Low Very Low
Central nervous system 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Stress 28 90.40 High High
Neuroblastoma 2 19.84 Low Low
Targeted Disruption 10 5.00 Very Low Very Low Very Low
Hyponatremia 5 5.00 Very Low Very Low Very Low
Anxiety Disorder 4 5.00 Very Low Very Low Very Low
INFLAMMATION 4 5.00 Very Low Very Low Very Low
Small Cell Lung Cancer 4 5.00 Very Low Very Low Very Low
Cancer 3 5.00 Very Low Very Low Very Low
Depression 2 5.00 Very Low Very Low Very Low
Adrenal Insufficiency 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The AP1 site we identified is the sole responsible element for Fos/Jun proteins, because the AP1-induced potent transcription activity of the vasopressin gene is completely abolished when the AP1 site is altered.
Negative_regulation (abolished) of AP1 Positive_regulation (induced) of vasopressin gene
1) Confidence 0.05 Published 2008 Journal Gene Regulation and Systems Biology Section Body Doc Link PMC2733102 Disease Relevance 0.23 Pain Relevance 0.06

General Comments

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