INT281127

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Context Info
Confidence 0.08
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.42
Pain Relevance 0.30

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

endosome (Cxcr4) cytoplasmic membrane-bounded vesicle (Cxcr4) plasma membrane (Cxcr4)
nucleus (Tcf21) signal transducer activity (Cxcr4)
Anatomy Link Frequency
nerve 2
Tcf21 (Rattus norvegicus)
Cxcr4 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
dorsal root ganglion 42 99.40 Very High Very High Very High
Peripheral nerve injury 3 77.92 Quite High
chemokine 52 69.08 Quite High
Neuropathic pain 12 53.64 Quite High
painful pain 1 20.28 Low Low
antagonist 18 5.00 Very Low Very Low Very Low
withdrawal 12 5.00 Very Low Very Low Very Low
qutenza 7 5.00 Very Low Very Low Very Low
Sciatic nerve 6 5.00 Very Low Very Low Very Low
Pain 6 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Ganglion Cysts 42 99.40 Very High Very High Very High
Nervous System Injury 8 77.92 Quite High
Injury 33 63.32 Quite High
Neuropathic Pain 16 53.64 Quite High
Acquired Immune Deficiency Syndrome Or Hiv Infection 21 50.00 Quite Low
Nociception 7 20.28 Low Low
Neuropathy 13 5.00 Very Low Very Low Very Low
Rheumatoid Arthritis 10 5.00 Very Low Very Low Very Low
Hyperalgesia 5 5.00 Very Low Very Low Very Low
Peripheral Neuropathy 5 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In contrast, combined treatment of animals with gp120/hCD4 and ddC produced an increase in the number of neurons expressing CXCR4 protein by POD21 in the lumbar DRG ipsilateral to the nerve lesion.
POD21 Positive_regulation (increase) of Gene_expression (expressing) of CXCR4 protein in nerve associated with dorsal root ganglion
1) Confidence 0.08 Published 2009 Journal Mol Pain Section Body Doc Link PMC2734548 Disease Relevance 0.42 Pain Relevance 0.30

General Comments

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