INT281610

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.02
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.12
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

carbohydrate metabolic process (Akt1, Gsk3b) cytoplasm (Akt1, Gsk3b) cytosol (Akt1, Gsk3b)
nucleus (Akt1, Gsk3b) plasma membrane (Akt1, Gsk3b) kinase activity (Akt1, Gsk3b)
Akt1 (Rattus norvegicus)
Gsk3b (Rattus norvegicus)
Pain Link Frequency Relevance Heat
cytokine 3 5.00 Very Low Very Low Very Low
analgesia 1 5.00 Very Low Very Low Very Low
nalbuphine 1 5.00 Very Low Very Low Very Low
Spinal cord 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Sepsis 3 64.40 Quite High
Heart Rate Under Development 5 5.00 Very Low Very Low Very Low
Myocardial Infarction 2 5.00 Very Low Very Low Very Low
Coronary Heart Disease 1 5.00 Very Low Very Low Very Low
Muscular Atrophy 1 5.00 Very Low Very Low Very Low
Necrosis 1 5.00 Very Low Very Low Very Low
Cancer 1 5.00 Very Low Very Low Very Low
Pressure Volume 2 Under Development 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Indeed, AKT is well known as an upstream regulator of translation initiation, where increases in AKT activity lead to increases in both eIF2B guanidine exchange factor activity and eIF4F assembly, via the phosphorylation and subsequent inhibition of GSK3?
Positive_regulation (increases) of AKT Positive_regulation (increases) of factor
1) Confidence 0.02 Published 2009 Journal PLoS ONE Section Body Doc Link PMC2736646 Disease Relevance 0.12 Pain Relevance 0

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox