INT282090

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Context Info
Confidence 0.33
First Reported 2009
Last Reported 2010
Negated 1
Speculated 0
Reported most in Body
Documents 2
Total Number 2
Disease Relevance 2.02
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleoplasm (Cdkn2a) aging (Cdkn2a) DNA binding (Cdkn2a)
protein complex (Cdkn2a) cytoplasm (Cdkn2a) nucleolus (Cdkn2a)
Cdkn2a (Mus musculus)
Pain Link Frequency Relevance Heat
palliative 1 5.00 Very Low Very Low Very Low
metalloproteinase 1 5.00 Very Low Very Low Very Low
fibrosis 1 5.00 Very Low Very Low Very Low
Immobilon 1 5.00 Very Low Very Low Very Low
Dismenorea 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Nasopharynx Cancer 53 97.28 Very High Very High Very High
Aging 24 92.72 High High
Congenital Anomalies 1 90.88 High High
Epstein-barr Virus 6 89.00 High High
Genetic Predisposition To Disease 1 88.80 High High
Infection 1 87.72 High High
Cancer 54 83.96 Quite High
Lung Cancer 1 76.56 Quite High
Colon Cancer 2 65.24 Quite High
Stress 3 63.24 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Western blotting for P53, P21 and P16 proteins revealed a rapid enhancement of P53 protein followed by an increased in P21 levels, while P16 levels remained unchanged (Figure 7b).
Neg (unchanged) Regulation (unchanged) of P16
1) Confidence 0.33 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2884035 Disease Relevance 0.59 Pain Relevance 0
Besides the LMP1-induced oncogenic pathways, dysregulation of factors such as p16, cyclin D1, and cyclin E leads to aberrations in the cell cycle in NPC cells.
Regulation (dysregulation) of p16 associated with nasopharynx cancer
2) Confidence 0.08 Published 2009 Journal J Transl Med Section Body Doc Link PMC2742515 Disease Relevance 1.43 Pain Relevance 0

General Comments

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