INT282950

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Context Info
Confidence 0.05
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.05
Pain Relevance 0.03

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (UBA7) protein modification process (UBA7) ligase activity (UBA7)
Anatomy Link Frequency
neural 1
UBA7 (Homo sapiens)
Pain Link Frequency Relevance Heat
antagonist 3 68.40 Quite High
nMDA receptor 3 5.00 Very Low Very Low Very Low
Glutamate receptor 3 5.00 Very Low Very Low Very Low
agonist 3 5.00 Very Low Very Low Very Low
imagery 3 5.00 Very Low Very Low Very Low
Action potential 2 5.00 Very Low Very Low Very Low
tetrodotoxin 1 5.00 Very Low Very Low Very Low
Spinal cord 1 5.00 Very Low Very Low Very Low
gABA 1 5.00 Very Low Very Low Very Low
Glutamate 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Neurodegenerative Disease 4 50.20 Quite High
Disease 1 39.84 Quite Low
Leukemia 1 5.00 Very Low Very Low Very Low
Peripheral Arterial Disease 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Under control conditions, hESCs can be readily converted to neural cells over 16 days (Fig. 1A) with concomitant loss of pluripotency markers OCT4 and NANOG, and up regulation of neural progenitor markers MUSASHI (D8) and SOX1 (D 16).
Regulation (regulation) of D8 in neural
1) Confidence 0.05 Published 2009 Journal PLoS ONE Section Body Doc Link PMC2752165 Disease Relevance 0.05 Pain Relevance 0.03

General Comments

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