INT282953

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.03
First Reported 2009
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 2
Disease Relevance 1.63
Pain Relevance 0.03

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

RNA binding (MSI1) nucleus (MSI1) cytoplasm (MSI1)
Anatomy Link Frequency
neural 1
MSI1 (Homo sapiens)
Pain Link Frequency Relevance Heat
antagonist 4 68.40 Quite High
nMDA receptor 3 5.00 Very Low Very Low Very Low
Glutamate receptor 3 5.00 Very Low Very Low Very Low
agonist 3 5.00 Very Low Very Low Very Low
imagery 3 5.00 Very Low Very Low Very Low
Action potential 2 5.00 Very Low Very Low Very Low
tetrodotoxin 1 5.00 Very Low Very Low Very Low
Spinal cord 1 5.00 Very Low Very Low Very Low
metalloproteinase 1 5.00 Very Low Very Low Very Low
pain pelvic 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Endometrial Cancer 153 100.00 Very High Very High Very High
Microsatellite Instability 47 100.00 Very High Very High Very High
Aneuploidy 3 96.32 Very High Very High Very High
Chromosomal Instability 1 92.08 High High
Carcinoma 61 83.04 Quite High
Endometroid Carcinoma 21 71.68 Quite High
Neurodegenerative Disease 4 50.20 Quite High
Disease 8 39.84 Quite Low
Skin Cancer 1 34.44 Quite Low
Aggression 3 29.56 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Furthermore, none of the five main alterations of endometrioid endometrial carcinoma (mutations of PTEN, PIK3CA, KRAS, and CTNNB1 genes and MSI) plays a major role in non-endometrioid endometrial carcinoma.
Regulation (alterations) of MSI associated with microsatellite instability and endometrial cancer
1) Confidence 0.03 Published 2010 Journal Obstetrics and Gynecology International Section Body Doc Link PMC2846683 Disease Relevance 1.58 Pain Relevance 0
Under control conditions, hESCs can be readily converted to neural cells over 16 days (Fig. 1A) with concomitant loss of pluripotency markers OCT4 and NANOG, and up regulation of neural progenitor markers MUSASHI (D8) and SOX1 (D 16).
Regulation (regulation) of MUSASHI in neural
2) Confidence 0.01 Published 2009 Journal PLoS ONE Section Body Doc Link PMC2752165 Disease Relevance 0.05 Pain Relevance 0.03

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox