INT282976

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Context Info
Confidence 0.29
First Reported 2009
Last Reported 2009
Negated 1
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.76
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (Bcl2) cell morphogenesis (Bcl2) endoplasmic reticulum (Bcl2)
intracellular (Bcl2) cytoplasm (Bcl2) cell proliferation (Bcl2)
Bcl2 (Mus musculus)
Pain Link Frequency Relevance Heat
anesthesia 1 5.00 Very Low Very Low Very Low
halothane 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apoptosis 104 100.00 Very High Very High Very High
Infection 51 96.08 Very High Very High Very High
Acquired Immune Deficiency Syndrome Or Hiv Infection 1 50.00 Quite Low
Viral Infection 13 5.00 Very Low Very Low Very Low
Sprains And Strains 4 5.00 Very Low Very Low Very Low
Death 3 5.00 Very Low Very Low Very Low
Starvation 3 5.00 Very Low Very Low Very Low
Stress 2 5.00 Very Low Very Low Very Low
Disease 2 5.00 Very Low Very Low Very Low
Toxicity 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Since the BH2 domain is involved in the ability of vBcl-2 to inhibit apoptosis but not autophagy, this result thus suggests that the inhibition of host apoptosis by vBcl-2 is required for efficient ex vivo reactivation from the latent state particularly at later time points after infection, which is consistent with the previous report that the ?
Positive_regulation (required) of vBcl-2 Neg (inhibition) Binding (inhibition) of associated with apoptosis and infection
1) Confidence 0.29 Published 2009 Journal PLoS Pathogens Section Body Doc Link PMC2752191 Disease Relevance 0.76 Pain Relevance 0

General Comments

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