INT284871

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Context Info
Confidence 0.44
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.27
Pain Relevance 0.04

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transport (Scn1b)
Anatomy Link Frequency
ventricles 1
Scn1b (Mus musculus)
Pain Link Frequency Relevance Heat
Action potential 26 79.28 Quite High
addiction 5 5.00 Very Low Very Low Very Low
action potential duration 5 5.00 Very Low Very Low Very Low
Eae 3 5.00 Very Low Very Low Very Low
Nav1.1 2 5.00 Very Low Very Low Very Low
sodium channel 1 5.00 Very Low Very Low Very Low
Nav1.9 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Congenital Anomalies 2 85.44 High High
Targeted Disruption 6 75.00 Quite High
Ventricular Tachycardia 22 57.76 Quite High
Arrhythmogenic Right Ventricular Dysplasia 13 5.00 Very Low Very Low Very Low
Heart Arrhythmia 9 5.00 Very Low Very Low Very Low
Syndrome 4 5.00 Very Low Very Low Very Low
Body Weight 3 5.00 Very Low Very Low Very Low
Camurati-engelmann Disease 2 5.00 Very Low Very Low Very Low
Adhesions 2 5.00 Very Low Very Low Very Low
Bartter Syndrome 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
These hearts also showed increased expression levels of Scn1b mRNA in both ventricles and Scn5a mRNA in the right ventricles compared to findings in WT hearts.
Positive_regulation (increased) of Scn1b in ventricles
1) Confidence 0.44 Published 2008 Journal Prog Biophys Mol Biol Section Abstract Doc Link PMC2764399 Disease Relevance 0.27 Pain Relevance 0.04

General Comments

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