INT285212

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Context Info
Confidence 0.30
First Reported 2009
Last Reported 2010
Negated 0
Speculated 1
Reported most in Body
Documents 1
Total Number 3
Disease Relevance 0.17
Pain Relevance 0.10

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

lyase activity (Adcy3) plasma membrane (Adcy3) nucleus (Adcy3)
cytoplasm (Adcy3)
Adcy3 (Mus musculus)
Pain Link Frequency Relevance Heat
lidocaine 6 71.52 Quite High
antagonist 22 64.84 Quite High
fibrosis 2 38.72 Quite Low
Kinase C 22 5.00 Very Low Very Low Very Low
adenocard 4 5.00 Very Low Very Low Very Low
endometriosis 2 5.00 Very Low Very Low Very Low
Dismenorea 2 5.00 Very Low Very Low Very Low
imagery 2 5.00 Very Low Very Low Very Low
ketamine 1 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Frailty 1 94.12 High High
Injury 2 80.12 Quite High
Fibrosis 2 38.72 Quite Low
Endometriosis (extended) 12 5.32 Low Low
Loss Of Sense Of Smell 30 5.00 Very Low Very Low Very Low
Necrosis 9 5.00 Very Low Very Low Very Low
Targeted Disruption 6 5.00 Very Low Very Low Very Low
Death 4 5.00 Very Low Very Low Very Low
Intestinal Cancer 4 5.00 Very Low Very Low Very Low
Reprotox - General 1 4 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Furthermore, we examined the expression of AC3, ?
Spec (examined) Negative_regulation (examined) of Spec (examined) Gene_expression (expression) of AC3
1) Confidence 0.30 Published 2009 Journal PLoS ONE Section Body Doc Link PMC2765727 Disease Relevance 0.17 Pain Relevance 0.07
Three different Stealth siRNA duplex oligoribonucleotides (Invitrogen, Paisley, UK) were used to abolish the expression and function of AC1 and AC3.
Negative_regulation (abolish) of Gene_expression (expression) of AC3
2) Confidence 0.24 Published 2010 Journal Cell Signal Section Body Doc Link PMC2791881 Disease Relevance 0 Pain Relevance 0
Transfection studies using siRNA to abolish expression of AC1 or AC3 in FPEP2 cells revealed that the calcium sensitive AC3 (but not AC1) isoform is responsible for PGF-mediated potentiation of Butaprost-stimulated cAMP.
Negative_regulation (abolish) of Gene_expression (expression) of AC3
3) Confidence 0.24 Published 2010 Journal Cell Signal Section Body Doc Link PMC2791881 Disease Relevance 0 Pain Relevance 0.03

General Comments

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