INT285631

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Context Info
Confidence 0.31
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.41
Pain Relevance 0.12

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular region (Cd5l)
Anatomy Link Frequency
goblet cell 1
Cd5l (Mus musculus)
Pain Link Frequency Relevance Heat
Inflammation 14 98.14 Very High Very High Very High
metalloproteinase 1 87.28 High High
cytokine 7 5.00 Very Low Very Low Very Low
fibrosis 7 5.00 Very Low Very Low Very Low
Thoracotomy 6 5.00 Very Low Very Low Very Low
Inflammatory response 5 5.00 Very Low Very Low Very Low
ketamine 1 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
Buprenorphine 1 5.00 Very Low Very Low Very Low
Pain 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Fibrosis 17 98.34 Very High Very High Very High
INFLAMMATION 19 98.14 Very High Very High Very High
Hyperplasia 2 95.32 Very High Very High Very High
Disease 3 5.00 Very Low Very Low Very Low
Dislocations 2 5.00 Very Low Very Low Very Low
Injury 2 5.00 Very Low Very Low Very Low
Keloid Scars 2 5.00 Very Low Very Low Very Low
Stress 2 5.00 Very Low Very Low Very Low
Asthma 2 5.00 Very Low Very Low Very Low
Natriuresis 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Interestingly, genes relating to goblet cell hyperplasia and mucous production are also up-regulated (Clca3, Agr2, Slc26a4), with differential expression of other genes associated with inflammatory, fibrotic or immune responses (up-regulation of Reg3g, Ear11, Retnla, Nappa, and Ccr9; down-regulation of Arg1, CD5L and Alox15).
Negative_regulation (regulation) of CD5L in goblet cell associated with fibrosis, inflammation and hyperplasia
1) Confidence 0.31 Published 2009 Journal Respir Res Section Body Doc Link PMC2770038 Disease Relevance 0.41 Pain Relevance 0.12

General Comments

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