INT285646

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Context Info
Confidence 0.28
First Reported 2009
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 4
Total Number 4
Disease Relevance 1.01
Pain Relevance 1.84

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transcription factor binding (Naaa) biological_process (Naaa) lipid metabolic process (Naaa)
lysosome (Naaa) cytoplasm (Naaa)
Anatomy Link Frequency
brain 1
Naaa (Mus musculus)
Pain Link Frequency Relevance Heat
Endocannabinoid 132 97.56 Very High Very High Very High
Neuropathic pain 51 87.32 High High
Spinal cord 48 86.12 High High
Pain 51 74.92 Quite High
Spinal sensitization 3 73.60 Quite High
Peripheral nerve injury 3 57.00 Quite High
descending inhibitory control 3 46.08 Quite Low
substance P 3 42.92 Quite Low
Hippocampus 12 42.56 Quite Low
Periaqueductal grey 21 37.76 Quite Low
Disease Link Frequency Relevance Heat
Neuropathic Pain 87 87.32 High High
Pain 78 74.92 Quite High
Nervous System Injury 3 57.00 Quite High
Urological Neuroanatomy 21 37.76 Quite Low
Nociception 45 36.12 Quite Low
Impaired Glucose Tolerance 16 35.92 Quite Low
Inflammatory Pain 27 34.56 Quite Low
INFLAMMATION 33 33.20 Quite Low
Injury 24 30.56 Quite Low
Hyperalgesia 27 5.72 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Hydrolysing enzymes include fatty acid amide hydrolase (FAAH), monoacylglycerol lipase (MAGL) and N-acylethanolamine-hydrolysing acid amidase (NAAA).
Gene_expression (include) of NAAA
1) Confidence 0.28 Published 2009 Journal Mol Pain Section Body Doc Link PMC2770047 Disease Relevance 0.48 Pain Relevance 0.76
Hydrolysing enzymes include fatty acid amide hydrolase (FAAH), monoacylglycerol lipase (MAGL) and N-acylethanolamine-hydrolysing acid amidase (NAAA).
Gene_expression (include) of N-acylethanolamine-hydrolysing acid amidase
2) Confidence 0.28 Published 2009 Journal Mol Pain Section Body Doc Link PMC2770047 Disease Relevance 0.48 Pain Relevance 0.76
Levels of NAAA are low in the brain and the enzyme is unlikely to be an important mediator of endocannabinoid metabolism under normal conditions.
Gene_expression (Levels) of NAAA in brain associated with endocannabinoid
3) Confidence 0.25 Published 2009 Journal Mol Pain Section Body Doc Link PMC2770047 Disease Relevance 0.06 Pain Relevance 0.32
Enteroendocrine L cells contain PYY and express the acylethanolamine receptor, Gpr119.
Gene_expression (express) of acylethanolamine receptor
4) Confidence 0.01 Published 2010 Journal Cell Metabolism Section Abstract Doc Link PMC2890049 Disease Relevance 0 Pain Relevance 0

General Comments

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