INT285902

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Context Info
Confidence 0.06
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.60
Pain Relevance 0.13

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell differentiation (AKT1) nucleoplasm (AKT1) transport (AKT1)
small molecule metabolic process (AKT1) enzyme binding (AKT1) carbohydrate metabolic process (AKT1)
AKT1 (Homo sapiens)
Pain Link Frequency Relevance Heat
Inflammation 34 78.24 Quite High
Inflammatory mediators 7 71.64 Quite High
Central nervous system 15 36.88 Quite Low
Multiple sclerosis 2 10.00 Low Low
chemokine 18 6.60 Low Low
cytokine 17 5.00 Very Low Very Low Very Low
Inflammatory response 3 5.00 Very Low Very Low Very Low
Neuritis 1 5.00 Very Low Very Low Very Low
Inflammatory stimuli 1 5.00 Very Low Very Low Very Low
imagery 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
INFLAMMATION 45 78.24 Quite High
Apoptosis 56 74.48 Quite High
Lyme Disease 13 50.00 Quite Low
Multiple Sclerosis 5 15.44 Low Low
Cancer 1 13.68 Low Low
Neurodegenerative Disease 10 5.00 Very Low Very Low Very Low
Neuroblastoma 5 5.00 Very Low Very Low Very Low
Lyme Neuroborreliosis 5 5.00 Very Low Very Low Very Low
Infection 5 5.00 Very Low Very Low Very Low
Disease 3 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
This conclusion was founded on the observed upregulation in transcripts for AKT1 and Bcl-XL, paired with decreased gene expression for DRAM.
Positive_regulation (upregulation) of Transcription (transcripts) of AKT1
1) Confidence 0.06 Published 2009 Journal PLoS Pathogens Section Body Doc Link PMC2771360 Disease Relevance 0.60 Pain Relevance 0.13

General Comments

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