INT285982

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Context Info
Confidence 0.02
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 1.41
Pain Relevance 0.08

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell differentiation (Ros1) cell proliferation (Ros1) nucleolus (Trib3)
nucleus (Trib3) plasma membrane (Ros1) enzyme binding (Trib3)
Ros1 (Rattus norvegicus)
Trib3 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
ischemia 21 82.72 Quite High
Inflammation 4 5.00 Very Low Very Low Very Low
imagery 2 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apoptosis 45 99.48 Very High Very High Very High
Myocardial Reperfusion Injury 12 99.46 Very High Very High Very High
Death 4 85.24 High High
Cv Unclassified Under Development 10 82.72 Quite High
Necrosis 5 78.52 Quite High
Reperfusion Injury 12 77.44 Quite High
Myocardial Infarction 6 64.60 Quite High
Injury 15 5.00 Very Low Very Low Very Low
Coronary Artery Disease 11 5.00 Very Low Very Low Very Low
INFLAMMATION 4 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
However, reactive oxygen species (ROS) are formed in excessive amounts within the first few minutes following reperfusion and are considered important to myocardial reperfusion injury.5) In addition, excessive ROS during the early stages of reperfusion increases the activity of mitogen-activated protein (MAP) kinase and induces apoptosis following reperfusion.6-8)
ROS Positive_regulation (increases) of kinase Binding (activity) of associated with myocardial reperfusion injury and apoptosis
1) Confidence 0.02 Published 2009 Journal Korean Circulation Journal Section Body Doc Link PMC2771824 Disease Relevance 1.41 Pain Relevance 0.08

General Comments

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