INT286105

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Context Info
Confidence 0.36
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 1.94
Pain Relevance 0.04

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (Mutyh) hydrolase activity, acting on glycosyl bonds (Mutyh) nucleus (Mutyh)
Anatomy Link Frequency
germline 1
Mutyh (Mus musculus)
Pain Link Frequency Relevance Heat
adenocard 2 78.80 Quite High
imagery 10 5.00 Very Low Very Low Very Low
Bile 8 5.00 Very Low Very Low Very Low
Central nervous system 6 5.00 Very Low Very Low Very Low
abdominal pain 6 5.00 Very Low Very Low Very Low
COX-2 inhibitor 4 5.00 Very Low Very Low Very Low
methotrexate 2 5.00 Very Low Very Low Very Low
fibrosis 2 5.00 Very Low Very Low Very Low
Dismenorea 2 5.00 Very Low Very Low Very Low
vincristine 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Familial Adenomatous Polyposis 480 99.90 Very High Very High Very High
Polyps 136 96.48 Very High Very High Very High
Adenoma 116 96.48 Very High Very High Very High
Hyperplasia 6 96.20 Very High Very High Very High
Cancer 132 90.72 High High
Colorectal Cancer 2 81.20 Quite High
Adenocarcinoma 18 78.64 Quite High
Colon Cancer 66 77.04 Quite High
Colonic Polyps 2 52.40 Quite High
Hereditary Nonpolyposis Colorectal Neoplasms 8 10.44 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Genetic testing for MUTYH mutation has been recommended for all patients who have tens to hundreds of colorectal adenomas with no identified germline mutation in the APC gene and with a family history compatible with an autosomal recessive mode of inheritance.
Positive_regulation (testing) of MUTYH in germline associated with adenoma and familial adenomatous polyposis
1) Confidence 0.36 Published 2009 Journal Orphanet J Rare Dis Section Body Doc Link PMC2772987 Disease Relevance 1.47 Pain Relevance 0
It is recommended that patients, who have a recessive family history compatible with AFAP, be evaluated for a MUTYH mutation.
Positive_regulation (evaluated) of MUTYH associated with familial adenomatous polyposis
2) Confidence 0.36 Published 2009 Journal Orphanet J Rare Dis Section Body Doc Link PMC2772987 Disease Relevance 0.47 Pain Relevance 0.04

General Comments

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